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Group 1 had 18 patients and a mean /- SD follow-up of 45.2 mupirocin antibiotic dimyristoylglycerol. Long-term oral Acyclovir / Aciclovir use seems to remain effective

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in decreasing the number of ocular herpes simplex virus recurrences beyond 12 months.. In group 2, patients received the treatment for at least 18

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months. Acyclovir / Aciclovir diphosphate diacylglycerol prodrugs may be useful in treating TK-deficient mutant and wild-type strains of HSV. Acyclovir / Aciclovir monophosphate added to the culture medium (outside the cell) did not circumvent the Acyclovir / Aciclovir resistance of the TK-negative DM21 mutant, presumably due to its conversion to Acyclovir / Aciclovir by phosphatases. We compared recurrences when both groups were using Acyclovir / Aciclovir (period 1) and when only group 2 was receiving the drug (period 2). Mean /- SD recurrence-free survival in period 2 was 15.3 /- 5.5 months in group 1 and 37.3 /- 6.3 months in group 2 (P .001). To restore Acyclovir / Aciclovir efficacy, we synthesized Acyclovir / Aciclovir diphosphate dimyristoylglycerol, an analog of a naturally occurring phospholipid, CDP-diacylglycerol. Acyclovir / Aciclovir diphosphate dimyristoylglycerol has substantial activity in DM21-infected cells (IC50 0.25 microM), whereas Acyclovir / Aciclovir and Acyclovir / Aciclovir monophosphate were ineffective (IC50 >

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100 microM).

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A phospholipid prodrug with activity against Acyclovir / Aciclovir-resistant herpes simplex fection with herpes simplex viruses (HSVs) resistant to treatment with Acyclovir / Aciclovir (9-[(2-hydroxyethoxy)-methyl]guanine, Zovirax) is a growing clinical problem in patients with AIDS and other immunosuppressed

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states. Long-term Acyclovir / Aciclovir use to prevent recurrent ocular herpes simplex virus infection.OBJECTIVE. Statistical analysis was performed with the t test, chi2 test, and Kaplan-Meier method. The phospholipid prodrug is active by means of

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TK-independent metabolic pathways that liberate Acyclovir / Aciclovir monophosphate inside the host cell. To evaluate the effectiveness of more than 12 months of oral Acyclovir / Aciclovir therapy in reducing recurrences of ocular herpes simplex virus. We retrospectively compared ocular herpes

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simplex virus recurrence in 2 groups of patients. Acyclovir / Aciclovir phosphates were 56 times greater in WI38 human lung fibroblasts incubated for 24 hr with [8-3H]Acyclovir / Aciclovir diphosphate dimyristoylglycerol

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relative to Acyclovir / Aciclovir. Group 2 had 22 patients and a mean /- SD follow-up of 42.4 /- 30.2 months. Similar results were obtained in TK-altered and TK-deficient strains of HSV-1 and in

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Acyclovir / Aciclovir-resistant isolates of HSV-2 obtained from two AIDS patients. In group 1, patients used oral Acyclovir / Aciclovir for at least 12 months and then discontinued the treatment. Six patients (33%) in group 1 and 4 patients (18%) in group 2 had recurrence in period 1 (P .3). Most virus isolates resistant to Acyclovir / Aciclovir are deficient or defective in virally coded thymidine kinase (TK), which converts Acyclovir / Aciclovir to Acyclovir / Aciclovir monophosphate in virus-infected cells.